Electrolyte Content of Blister Fluid in Pemphigus
نویسندگان
چکیده
منابع مشابه
Signaling Dependent and Independent Mechanisms in Pemphigus Vulgaris Blister Formation
Pemphigus vulgaris (PV) is an autoimmune epidermal blistering disease caused by autoantibodies directed against the desmosomal cadherin desmoglein-3 (Dsg3). Significant advances in our understanding of pemphigus pathomechanisms have been derived from the generation of pathogenic monoclonal Dsg3 antibodies. However, conflicting models for pemphigus pathogenicity have arisen from studies using ei...
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Pemphigus is a blistering disease characterized by pemphigus autoantibodies (PVIgG) directed mostly against desmogleins (Dsgs), resulting in the loss of keratinocyte adhesion (acantholysis). Yet, the mechanisms underlying blister formation remain to be clarified. We have shown previously that anti-Fas ligand (FasL) antibody (Ab) prevents PVIgG-induced caspase-8 activation and Dsg cleavage in hu...
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در این پایان نامه تاثیر دو نوع تست جزیی نگر و کلی نگر بر به یادسپاری محتوا ارزیابی شده که نتایج نشان دهندهکارایی تستهای کلی نگر بیشتر از سایر آزمونها است
15 صفحه اولFluid and Electrolyte Therapy
child with physical defects, rheumatic fever, tuberculosis, diabetes, obesity, glandular disturbances, and speech disorders. There are chapters on problems of children who are adopted, are illegitimate, or who belong to minority groups. There are sections and paragraphs which are well written and accurate. However, the book as a whole, in the opinion of this reviewer, is not likely to increase ...
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Background Stevens-Johnson syndrome (SJS) and toxic epidermal necrolysis (TEN) are life-threatening hypersensitivity reactions to medications characterized by keratinocyte apoptosis, the formation of subepidermal blisters, and skin detachment. Cytotoxic lymphocytes including CTLs and NK cells seem to be the main effectors of keratinocyte killing. Natural killer cytotoxic activity is regulated t...
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ژورنال
عنوان ژورنال: Journal of Investigative Dermatology
سال: 1942
ISSN: 0022-202X
DOI: 10.1038/jid.1942.45